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Other checkpoints in EAE pathogenesis have not been clearly defined, although multiple genetic loci are known to influence EAE development. Krishna Antioxidants Private Limited. Cells were pretreated with or without PD for 30 min and then treated with either TPA or phenylephrine for 10 min.
Journal of Lipid Research.
Calidad Auto Tech, Inc. GLUT1 is the principal isoform expressed in the fetal heart, tcs its expression is down-regulated following birth in normal myocardium 236concomitantly with the shift from glycolytic to oxidative metabolism 1. Enron Gas Liquids Incorporated. For Gasoline and Diesel Additives: We have used this model to investigate the regulation of GLUT1 expression in cardiac myocytes. These results suggest that PI3K activation contributes to the induction of the Glut1 promoter in response to TPA and phenylephrine and that it acts upstream of Ras.
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Kellogs’s Professional Products, Inc. In addition, active MAP kinase is required for induction of the c- fos and atrial natriuretic factor promoters by phenylephrine 4142and expression of constitutively active MEK1 results in overexpression of hypertrophic genes Kalk Holdings US, Inc.
We found that these mice remain resistant to EAE induction. Energy Alliance Technology Corporation. Fuel Activator Chemical Corporation.
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Cells were plated onto glass coverslips coated with gelatin and laminin and left untreated or treated with TPA or PE for 48 h. Chippewa Valley Ethanol Company. Advanced Fuel Services, Inc. Cardiac myocytes were transfected with a reporter construct encoding luciferase under the control of the Glut1 promoter. Bright Star Industries, Inc. Camin Cargo Control, Inc. Automotive Chemical Products, Inc. Adoptive transfer studies demonstrate that both T cells and non—T cells must express HSA in order for the pathogenic T cells to execute their effector function.
Figure 7 Ras activity is required for induction of the Glut1 promoter in myocytes. Contact Us Fuels and Fuel Additives. Bio Oil National Corporation.
The costs of publication of this article were defrayed in part by the payment of page charges. Basal glucose transport into cardiac myocytes is mediated by the GLUT1 isoform of glucose transporters, whereas the GLUT4 isoform is responsible for regulatable glucose transport. Hygeia Coca-Cola Bottling Company. Atlantic Pacific Automotive L. Figure 3 TPA and phenylephrine increase transcription from the Glut1 promoter. Elite Lubricant Research, Inc.
Green Plains Renewable Energy, Inc. ER expresses the kinase domain of Raf1 fused to the steroid-binding domain of the human estrogen receptor Kold Ban International, Ltd.
Global Fuel Technologies Inc. BP Amoco Chemical Company. Advanced Technology Lubricants, Inc.